Gut bacteria diversity is one of the key drivers of health.
The American Gut Project, and the Twins UK project, found that building diversity had a comparable effect to taking medication, and low diversity to disease. It’s that important.
Within the functional health community, there is a lot of focus on killing pathogens, viruses, and other bad guys. Many functional tests are aimed at identifying them – such as the Gi-Map™ test.
However, less emphasis has been placed on rebuilding or maintaining gut bacteria diversity – which the Ubiome Explorer test measures.
In my experience, you need both, otherwise, health is never quite restored. You are either busy shoveling the water out or busy trying to plug the holes in a leaky gut and biome.
One of the challenges faced by people with histamine intolerance is the removal of high histamine ferments, but fortunately, a wide range of low histamine foods nourish the gut. You do not need to eat sauerkraut to have a good gut biome.
A recent study has provided important information on diamine oxidase foods and how to raise diamine oxidase naturally through our diet.
The study looked at both macronutrients (protein, fat, and carbohydrates) and micronutrients (including key minerals and vitamins) in healthy women.
The findings suggest that our diet (and not just the histamine content of food) has a profound impact on our histamine degrading enzyme diamine oxidase. Increasing our diamine oxidase may help increase our histamine tolerance.
Australian scientists have linked chronic fatigue syndrome (CFS) to a genetic defect in immune cell receptors putting to rest once and for all the pathology of this much-maligned condition.
The research undertaken by the National Centre for Neuroimmunology and Emerging Diseases (Griffiths University) found that an inherited genetic defect means that cells do not work optimally.
These findings have potentially wider implications as many of the patients studied also had fibromyalgia and postural orthostatic tachycardia syndrome.
Mast-cell precursors have also been found in the blood of the patients studied. Whilst CFS patients might well fall under the spectrum of mast-cell activation, the Griffiths University research, by identifying the mechanism of action, provides a much more precise diagnosis.